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3586 16153 Ensembl ENSG00000136634 ENSMUSG00000016529 UniProt P22301 P18893 RefSeq (mRNA) NM_000572 NM_010548 RefSeq (protein) NP_000563 NP_034678 Location (UCSC) Chr 1: 206.77 – 206.77 Mb Chr 1: 130.95 – 130.95 Mb PubMed search Wikidata View/Edit Human View/Edit Mouse Interleukin 10 (IL-10), also known as human cytokine synthesis inhibitory factor (CSIF), is an anti- inflammatory cytokine ...
Cellular immunity, also known as cell-mediated immunity, is an immune response that does not rely on the production of antibodies. Rather, cell-mediated immunity is the activation of phagocytes , antigen-specific cytotoxic T-lymphocytes , and the release of various cytokines in response to an antigen .
He earned a Ph.D. from The Rockefeller University in 1981 in cellular immunology, working in Zanvil A. Cohn’s laboratory with Ralph M. Steinman on groundbreaking studies of mouse dendritic cells. As a Ph.D. student, Nussenzweig was the first to show that dendritic cells present foreign antigens to initiate T cell immunity. [ 1 ]
The first major immunologic model was the Self/Non-self Model proposed by Macfarlane Burnet and Frank Fenner in 1949 with later refinement by Burnet. [1] [2] It theorizes that the immune system distinguishes between self, which is tolerated, and non-self, which is attacked and destroyed.
[3] [2] [4] He is senior editor of the pathology reference book Robbins and Cotran Pathologic Basis of Disease along with Vinay Kumar, [5] as well as Basic Immunology, [6] and Cellular & Molecular Immunology. [7] [3] He was editor for Immunity from 1993 to 1996, and continues to serve as a member of the editorial board. [8]
The side-chain theory (German, Seitenkettentheorie) is a theory proposed by Paul Ehrlich (1854–1915) to explain the immune response in living cells.Ehrlich theorized from very early in his career that chemical structure could be used to explain why the immune response occurred in reaction to infection.
CD28 (Cluster of Differentiation 28) is a protein expressed on T cells that provides essential co-stimulatory signals required for T cell activation and survival. When T cells are stimulated through CD28 in conjunction with the T-cell receptor (), it enhances the production of various interleukins, particularly IL-6.
As of 2024, she is an emeritus professor at Scripps Research, and serves on the advisory boards of the La Jolla Institute for Immunology and the San Diego Biomedical Research Institute. [ 3 ] [ 4 ] Her research has focused on immune tolerance and autoimmunity , particularly type 1 diabetes , as well as the immune response to tumors .
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