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If a temporary crown becomes de-cemented, it is important that a dentist examine the patient as overeruption of the opposing teeth may prevent accurate fitting of the final crown. [8] If a dentist cannot be seen in a timely manner, the temporary crown may be re-cemented by applying temporary cement to the temporary crown.
Temporary crowns can either be direct, if constructed by the dentist in the clinic, or indirect if they are made off-site, usually in a dental laboratory. Generally direct temporary crowns tend to be for short-term use. Where medium-term or long-term temporisation is required, the use of indirect temporary crowns should be considered. [10]
Pericoronitis is inflammation of the soft tissues surrounding the crown of a partially erupted tooth, [1] including the gingiva (gums) and the dental follicle. [2] The soft tissue covering a partially erupted tooth is known as an operculum, an area which can be difficult to access with normal oral hygiene methods.
Gingival recession, also known as gum recession and receding gums, is the exposure in the roots of the teeth caused by a loss of gum tissue and/or retraction of the gingival margin from the crown of the teeth. [1] Gum recession is a common problem in adults over the age of 40, but it may also occur starting in adolescence, or around the age of 10.
Temporary restoration is a temporary filling of a prepared tooth until permanent restoration is carried out. It is used to cover the prepared part of the tooth, in order to maintain the occlusal space and the contact points, and insulation of the pulpal tissues and maintenance of the periodontal relationship.
It is a likely outcome of untreated dental caries (tooth decay), and in such cases it can be considered a sequela in the natural history of tooth decay, irreversible pulpitis and pulpal necrosis. Other causes can include occlusal trauma due to 'high spots' after restoration work, extrusion from the tooth of root filling material, or bacterial ...
Temporary filling-materials allow the creation of hermetic coronal-seals preventing from coronal microleakage (i.e. contamination of the root canal by bacteria); their presence over the entire time-period to fill the root canal and restore the tooth crown is mandatory, for increasing the probability of the endodontic-treatment success.
In 1890, W.D. Miller, considered the father of oral microbiology, was the first to associate pulpal disease with the presence of bacteria. [11] This was confirmed by Kakehashi, who, in 1965, proved that bacteria were the cause of pulpal and periradicular disease in studies using animal models; pulpal exposures were initiated in both normal and germ-free rats, and while no pathologic changes ...
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