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Bradycardia: The heart rate decreases significantly when the face is exposed to cold water. This helps to conserve oxygen by slowing down the heartbeat. The degree of bradycardia can vary among individuals, but it is a common and well-documented response.
Hypothermia is the cause of at least 1,500 deaths a year in the United States. [2] It is more common in older people and males. [ 5 ] One of the lowest documented body temperatures from which someone with accidental hypothermia has survived is 12.7 °C (54.9 °F) in a 2-year-old boy from Poland named Adam. [ 6 ]
Hypothermia is a low core body temperature, defined clinically as a temperature of less than 35 degrees Celsius (95 degrees Fahrenheit). The patient is re-warmed either by using a cardiac bypass or by irrigation of the body cavities (such as thorax, peritoneum, bladder) with warm fluids; or warmed IV fluids.
Severe hypothermia begins when the core body temperature is 82 degrees or lower, according to Huang. Symptoms include bluish skin, dilated pupils, slowed pulse and breathing, low blood pressure ...
Severe cases can cause cardiac arrest. [9] Hypothermia should be treated first, if present, by bringing core body temperature above 35 degrees Celsius. [6] [10] Raynaud's phenomenon: An abnormal spasming of blood vessels often in the tips of fingers and toes - usually in response to strong emotions or cold exposure. [8]
Bradycardia arrhythmia may have many causes, both cardiac and non-cardiac. Non-cardiac causes are usually secondary and can involve recreational drug use or abuse , metabolic or endocrine issues, especially hypothyroidism , an electrolyte imbalance , neurological factors, autonomic reflexes , situational factors, such as prolonged bed rest ...
Neurogenic shock is a distributive type of shock resulting in hypotension (low blood pressure), often with bradycardia (slowed heart rate), caused by disruption of autonomic nervous system pathways. [1] It can occur after damage to the central nervous system, such as spinal cord injury and traumatic brain injury.
The simultaneous loss of coagulation factors via hemorrhage, hemodilution with resuscitation fluids, and coagulation cascade dysfunction secondary to acidosis and hypothermia have been traditionally thought to be the cause of coagulopathy in trauma. However, this traditional model of trauma-induced coagulopathy may be too limited.