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A scaphoid fracture is a break of the scaphoid bone in the wrist. [1] ... If x-ray results show arthritis due to an old break, the treatment plan will first focus on ...
This burst causes the scaphoid bone to dorsally rotate. [3] A gap of more than 3mm is pathognomonic for scapholunate dissociation. [4] The resulting separation between the scaphoid and lunate bones leaves a space on the x-ray that is similar to the gap comedian Terry-Thomas had between his front teeth.
When the X-ray is diagnostic and there is a convincing Terry Thomas sign it is a static scaphoid instability. When the scaphoid is made unstable by either the patient or by manipulation by the examining physician it is a dynamic instability. [11] SNAC. In order to diagnose a SNAC wrist you need a PA view X-ray and a lateral view X-ray.
Healing of the fracture with a non-anatomic deformity (frequently, a volar flexed "humpback") can also lead to post-traumatic arthritis. Non-unions can result in loss of blood supply to the proximal pole, which can result in avascular necrosis of the proximal segment. Scaphoid fractures may be difficult to diagnose via plain x-ray.
Chauffeur's fracture, also known as Hutchinson fracture, is a type of intraarticular oblique fracture of the radial styloid process in the forearm. [1] The injury is typically caused by compression of the scaphoid bone of the hand against the styloid process of the distal radius. It can be caused by falling onto an outstretched hand.
The differential diagnosis includes scaphoid fractures and wrist dislocations, which can also co-exist with a distal radius fracture. Occasionally, fractures may not be seen on X-rays immediately after the injury. Delayed X-rays, X-ray computed tomography (CT scan), or Magnetic resonance imaging (MRI) can confirm the diagnosis. [citation needed]
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Preiser disease, or avascular necrosis of the scaphoid, is a rare condition where ischemia and necrosis of the scaphoid bone occurs without previous fracture.It is thought to be caused by repetitive microtrauma or side effects of drugs (e.g., steroids or chemotherapy) in conjunction with existing defective vascular supply to the proximal pole of the scaphoid.