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Orbital cellulitis is inflammation of eye tissues behind the orbital septum. It is most commonly caused by an acute spread of infection into the eye socket from either the adjacent sinuses or through the blood. It may also occur after trauma. When it affects the rear of the eye, it is known as retro-orbital cellulitis.
Ocular prothesis can mitigate symptoms of physiological distress related to phantom eye syndrome through simulating the functions of a normal eye socket. [4] Overall, the literature promotes treatment that focuses on increasing quality of life through addressing personalized emotional and physical phantom eye syndrome stressors. [4]
The practice is promoted by advocates as causing rapid intoxication, but the amount of alcohol absorbed by the eye is low. [ 8 ] Some observers maintained that the phenomenon was not a real craze, describing the coverage as a media feeding frenzy and part of "a long history of trend pieces that come out of nowhere".
An orbital blowout fracture is a traumatic deformity of the orbital floor or medial wall that typically results from the impact of a blunt object larger than the orbital aperture, or eye socket. [1] Most commonly this results in a herniation of orbital contents through the orbital fractures. [ 1 ]
SCS presents in a variable fashion. The majority of individuals with SCS are moderately affected, with uneven facial features and a relatively flat face due to underdeveloped eye sockets, cheekbones, and lower jaw. In addition to the physical abnormalities, people with SCS also experience growth delays, which results in a relatively short stature.
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There are two important foramina, or windows, two important fissures, or grooves, and one canal surrounding the globe in the orbit. There is a supraorbital foramen, an infraorbital foramen, a superior orbital fissure, an inferior orbital fissure and the optic canal, each of which contains structures that are crucial to normal eye functioning.