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[nb 3] [23] A shunting of blood from the thigh veins back into the lower-leg veins produces a reflux situation. The veins most often found to be incompetent are the saphenous veins and the perforators communicating with the deep veins of the thigh.
Chronic venous insufficiency (CVI) is a medical condition characterized by blood pooling in the veins, leading to increased pressure and strain on the vein walls. [1] The most common cause of CVI is superficial venous reflux, which often results in the formation of varicose veins, a treatable condition. [2]
Superficial veins are veins that are close to the surface of the body, as opposed to deep veins, which are far from the surface. Superficial veins are not paired with an artery, unlike the deep veins, which are typically associated with an artery of the same name. Superficial veins are important physiologically for cooling of the
Diagram showing named veins. Perforator veins exist along the length of the lower limb, in greater number in the leg (anatomical ref to below knee) than in the thigh. Some veins are named after the physician who first described them: Dodd's perforator at the inferior 1/3 of the thigh; Boyd's perforator at the knee level
The great saphenous vein (GSV) or long saphenous vein (/ s ə ˈ f iː n ə s /) is a large, subcutaneous, superficial vein of the leg. It is the longest vein in the body, running along the length of the lower limb, returning blood from the foot , leg and thigh to the deep femoral vein at the femoral triangle .
A long, thin red area may be seen as the inflammation follows a superficial vein. This area may feel hard, warm, and tender. The skin around the vein may be itchy and swollen. The area may begin to throb or burn. Symptoms may be worse when the leg is lowered, especially when first getting out of bed in the morning. A low-grade fever may occur ...
Venous stasis results from damage to the vein valvular system in the lower extremity and in extreme cases allows the pressure in the veins to be higher than the pressure in the arteries. This pressure results in transudation of inflammatory mediators into the subcutaneous tissues of the lower extremity and subsequent breakdown of the tissue ...
Vascular (or arterial) claudication typically occurs after activity or ambulation for a distance with resultant vascular insufficiency (lack of blood flow) where the muscular demands of oxygen outweighs the supply. Symptoms are lower extremity cramping. Resting from activity even in a standing position may help relieve the symptoms.