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Vasodilatory shock, vasogenic shock, or vasoplegic shock is a medical emergency belonging to shock along with cardiogenic shock, septic shock, allergen-induced shock and hypovolemic shock. When the blood vessels suddenly relax, it results in vasodilation. In vasodilatory shock, the blood vessels are too relaxed leading to extreme vasodilation ...
Vasodilation, also known as vasorelaxation, is the widening of blood vessels. [ 1 ] It results from relaxation of smooth muscle cells within the vessel walls, in particular in the large veins, large arteries, and smaller arterioles. [ 2 ] Blood vessel walls are composed of endothelial tissue and a basal membrane lining the lumen of the vessel ...
In physiology, acute local blood flow regulation refers to an intrinsic regulation, or control, of the vascular tone of arteries at a local level, meaning within a certain tissue type, organ, or organ system. This intrinsic type of control means that the blood vessels can automatically adjust their own vascular tone, by dilating (widening) or ...
Nitric oxide (nitrogen monoxide) is a molecule and chemical compound with chemical formula of N O. In mammals including humans, nitric oxide is a signaling molecule involved in several physiological and pathological processes. [1] It is a powerful vasodilator with a half-life of a few seconds in the blood.
Risk of death 20 to 50% [ 3 ] Frequency. 1.2 million per year (US) [ 3 ] Shock is the state of insufficient blood flow to the tissues of the body as a result of problems with the circulatory system. [ 1 ][ 2 ] Initial symptoms of shock may include weakness, fast heart rate, fast breathing, sweating, anxiety, and increased thirst. [ 1 ]
This area of the skin will become warmer because of vasodilation (loss of vasoconstriction). [5] Depression of the vasomotor center of the brain can cause the loss of vasomotor tone of blood vessels, resulting in massive dilatation of veins. This will result in a condition called as neurogenic shock. [6]
[6] [7] [8] VIP stimulates contractility in the heart, causes vasodilation, increases glycogenolysis, lowers arterial blood pressure and relaxes the smooth muscle of trachea, stomach and gallbladder. In humans, the vasoactive intestinal peptide is encoded by the VIP gene. [9] VIP has a half-life (t ½) in the blood of about two minutes. [10]
Endothelin functions through activation of two G protein-coupled receptors, endothelin A and endothelin B receptor (ETA and ETB, respectively). [2] These two subtypes of endothelin receptor are distinguished in the laboratory by the order of their affinity for the three endothelin peptides: the ETA receptor is selective for ET-1, whereas the ETB receptor has the same affinity for all three ET ...