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The frontal eye fields (FEF) are a region located in the frontal cortex, more specifically in Brodmann area 8 or BA8, [1] of the primate brain. In humans, it can be more accurately said to lie in a region around the intersection of the middle frontal gyrus with the precentral gyrus , consisting of a frontal and parietal portion. [ 2 ]
Visual field-bitemporal hemianopia Visual field-binasal hemianopia. A lesion involving complete optic chiasm, which disrupts the axons from the nasal field of both eyes, causes loss of vision of the right half of the right visual field and the left half of the left visual field. [3] This visual field defect is called as bitemporal hemianopia.
Brodmann area 8, or BA8, is part of the frontal cortex in the human brain. Situated just anterior to the premotor cortex , it includes the frontal eye fields (so-named because they are believed to play an important role in the control of eye movements). Damage to this area, by stroke, trauma or infection, causes tonic deviation of the eyes ...
Frontal eye field. The frontal eye field is a posterior part of the middle frontal gyrus and is involved in the control of saccadic, contralateral and conjugate eye movement. This area receives its main inputs from both the occipital cortex and dorsal thalamus. [6] Broca's area
Frontal release signs are primitive reflexes traditionally held to be a sign of disorders that affect the frontal lobes. The appearance of such signs reflects the area of brain dysfunction rather than a specific disorder which may be diffuse, such as a dementia, or localised, such as a tumor.
Signs and symptoms are classified into three groups based on the affected functions of the frontal and temporal lobes: [8] These are behavioural variant frontotemporal dementia, semantic dementia, and progressive nonfluent aphasia. An overlap between symptoms can occur as the disease progresses and spreads through the brain regions. [14]
Pontine lesions can typically be distinguished from supranuclear lesions in the frontal lobe based on clinical neurologic findings. Gaze palsies secondary to frontal lobe lesions can be temporarily relieved with rapid, passive horizontal head rotation, which also directly stimulates the sixth nerve nucleus through the vestibuloocular reflex .
In a 2006 study, patients with left prefrontal lesions on the SFG exhibited poorer results on working memory tasks than the control group. Mapping showed the lateral and posterior portions of the SFG contributed the most to the working memory deficit (mostly in Brodmann area 8 in front of the frontal eye field). This study suggests the SFG is ...