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In North America and Australia, DSM-5 criteria are used for diagnosis, while European countries usually use the ICD-10. The DSM-IV criteria for diagnosis of ADHD is 3–4 times more likely to diagnose ADHD than is the ICD-10 criteria. [210] ADHD is alternately classified as neurodevelopmental disorder [211] or a disruptive behaviour disorder ...
Animation of the frontal lobe, shown in red. Fronto-cerebellar dissociation is the disconnection and independent function of frontal and cerebellar regions of the brain.It is characterized by inhibited communication between the two regions, and is notably observed in cases of ADHD, schizophrenia, alcohol use disorder, and heroin use.
Simplified diagram of frontal cortex to striatum to thalamus pathways. There are five defined frontostriatal circuits: motor and oculomotor circuits originating in the frontal eye fields are involved in motor functions; while dorsolateral prefrontal, orbital frontal, and anterior cingulate circuits are involved in executive functions, social behavior and motivational states. [2]
ADHD can only be diagnosed by a licensed clinician, and the first step to do so is via screening with validated tools to screen for ADHD in adults. [10] [12] [13] The Adult ADHD Self Report Rating Scale (ASRS) is a validated screening tool recognized by the World Health Organization (WHO) with a sensitivity and specificity of 91.4% and 96.0% ...
The prefrontal cortex. Hypofrontality is a state of decreased cerebral blood flow (CBF) in the prefrontal cortex of the brain. Hypofrontality is symptomatic of several neurological medical conditions, such as schizophrenia, attention deficit hyperactivity disorder (ADHD), bipolar disorder, and major depressive disorder.
Frontal lobe disorder, also frontal lobe syndrome, is an impairment of the frontal lobe of the brain due to disease or frontal lobe injury. [5] The frontal lobe plays a key role in executive functions such as motivation, planning, social behaviour, and speech production.
In humans the frontal pole area of the prefrontal cortex includes not only area 10 but part of BA 9. BA 10 also extends beyond the pole area into its ventromedial side. In Guenon monkeys, the pole area is filled by BA 12 (and its BA 10 is found in the orbital prefrontal region). [2]
In a 2006 study, patients with left prefrontal lesions on the SFG exhibited poorer results on working memory tasks than the control group. Mapping showed the lateral and posterior portions of the SFG contributed the most to the working memory deficit (mostly in Brodmann area 8 in front of the frontal eye field ).