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Amyotrophic lateral sclerosis (ALS), also known as motor neuron disease (MND) or (in the United States) Lou Gehrig's disease (LGD), is a rare, terminal neurodegenerative disorder that results in the progressive loss of both upper and lower motor neurons that normally control voluntary muscle contraction. [3]
Neurodegenerative diseases include amyotrophic lateral sclerosis, multiple sclerosis, Parkinson's disease, Alzheimer's disease, Huntington's disease, multiple system atrophy, tauopathies, and prion diseases. Neurodegeneration can be found in the brain at many different levels of neuronal circuitry, ranging from molecular to systemic. [4]
Emotional disturbance (e.g. pseudobulbar affect) and cognitive and behavioural changes (e.g. problems in word fluency, decision-making, and memory) are also seen. [ 2 ] [ 6 ] There can be lower motor neuron findings (e.g. muscle wasting, muscle twitching), upper motor neuron findings (e.g. brisk reflexes, Babinski reflex , Hoffman's reflex ...
Mark, a Pennsylvania grandfather with ALS, is participating in a human trial with Synchron and is one of the first patients to be implanted with a brain-computer interface with the company. - CNN
Researchers identified a new biomarker for predicting the clinical outcome of patients with ALS, Amyotrophic Lateral Sclerosis, through brain imaging. Researchers Say This Test Can Predict ALS ...
The tech can't control your brain (at least not yet) — it does the opposite. But it could one day be used by the military, pilots, or even regular computer users.
Stem cell therapy can provide additional proteins and enzymes that have shown to help prolong survival and control the symptoms associated with ALS. [ 16 ] [ 17 ] Those proteins include neurotrophic factors and insulin-like growth factor 1.
FUS, which codes for "Fused in sarcoma" protein, is associated with 1–5% of familial ALS and less than 1% of sporadic ALS. FUS is an RNA-binding protein with a similar function to TDP-43. [6] Some people have both ALS and frontotemporal dementia (FTD–ALS). The four main genes associated with FTD–ALS are C9orf72, CHCHD10, SQSTM1, and TBK1. [8]