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A posterior vitreous detachment (PVD) is a condition of the eye in which the vitreous membrane separates from the retina. [1] It refers to the separation of the posterior hyaloid membrane from the retina anywhere posterior to the vitreous base (a 3–4 mm wide attachment to the ora serrata ).
The most common cause found in adults is diabetic retinopathy. Abnormal blood vessels can form in the back of the eye of a person with diabetes. These new blood vessels are weaker and prone to breaking and causing hemorrhage. [2] Diabetic retinopathy accounts for 31.5–54% of all cases of vitreous hemorrhage in adults in the United States. [1]
Epiretinal membrane or macular pucker is a disease of the eye in response to changes in the vitreous humor or more rarely, diabetes.Sometimes, as a result of immune system response to protect the retina, cells converge in the macular area as the vitreous ages and pulls away in posterior vitreous detachment (PVD).
If an identifiable disease does not cause this, it is called a primary vascular dysregulation (PVD); in case of an underlying disease, it is called secondary PVD. PVD is almost always associated with other vascular and non-vascular symptoms and signs. [19] This entire complex (PVD and accompanying symptoms) is today called Flammer syndrome. [20]
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Peripheral vascular disease, or PVD usually involves the arteries, and so sometimes it’s also referred to as peripheral artery disease or PAD. PVD is most often caused by a blockage, called organic PVD, most commonly caused by atherosclerosis, which is a buildup of lipids and fibrous material just under the inner lining of the blood vessel ...
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