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Pulseless electrical activity (PEA) is a form of cardiac arrest in which the electrocardiogram shows a heart rhythm that should produce a pulse, but does not.Pulseless electrical activity is found initially in about 20% of out-of-hospital cardiac arrests [1] and about 50% of in-hospital cardiac arrests.
While a heart attack refers to death of heart muscle tissue as a result of blood supply loss, cardiac arrest is caused when the heart's electrical system malfunctions. Furthermore, the American Heart Association explains that "if corrective measures are not taken rapidly, this condition progresses to sudden death.
Congenital heart defects are structural or electrical pathway problems in the heart that are present at birth. Anyone can be affected by this because overall health does not play a role in the problem. Problems with the electrical pathway of the heart can cause very fast or even deadly arrhythmias.
Drawing of the ECG, with labels of intervals. Cardiac electrophysiology is a branch of cardiology and basic science focusing on the electrical activities of the heart.The term is usually used in clinical context, to describe studies of such phenomena by invasive (intracardiac) catheter recording of spontaneous activity as well as of cardiac responses to programmed electrical stimulation ...
A cardiac electrophysiology study (EP test or EP study) is a minimally invasive procedure using catheters introduced through a vein or artery to record electrical activity from within the heart. [1] This electrical activity is recorded when the heart is in a normal rhythm (sinus rhythm) to assess the conduction system of the heart and to look ...
The most effective treatment to terminate torsades is an electrical cardioversion – a procedure in which an electrical current is applied across the heart to temporarily stop and then resynchronise the heart's cells. [23] Treatment to prevent recurrent torsades includes infusion of magnesium sulphate, [24] correction of electrolyte imbalances ...
Synchronized electrical cardioversion is used to treat hemodynamically unstable supraventricular (or narrow complex) tachycardias, including atrial fibrillation and atrial flutter. It is also used in the emergent treatment of wide complex tachycardias, including ventricular tachycardia, when a pulse is present.
Afterdepolarization is commonly a consequence of myocardial infarction, cardiac hypertrophy, or heart failure. [1] It may also result from congenital mutations associated with calcium channels and sequestration. [2]
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