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There are many causes that can induce or contribute to the development of bronchiectasis. The frequency of these different causes varies with geographic location. [24] Cystic fibrosis is identified as a cause in up to half of cases. [3] Bronchiectasis without CF is known as non-CF bronchiectasis.
[4] [5] [6] Due to its rarity it presents a difficulty in adult diagnoses, and its initial presentation can be confused with septic shock. [ 7 ] Diagnosis requires an appropriate clinical history , the characteristic expiratory airway collapse on radiological investigation, and exclusion of other causes of congenital and acquired bronchiectasis .
Respiratory syncytial virus (RSV), [a] also called human respiratory syncytial virus (hRSV) and human orthopneumovirus, is a virus that causes infections of the respiratory tract. It is a negative-sense, single-stranded RNA virus. [2] Its name is derived from the large cells known as syncytia that form when infected cells fuse. [2] [3]
The global under-five mortality rate in 1950 was 22.5%, which dropped to 4.5% in 2015. [10] Over the same period, the infant mortality rate declined from 65 deaths per 1,000 live births to 29 deaths per 1,000. [12] Globally, 5.4 million children died before their fifth birthday in 2017; [13] by 2021 that number had dropped to 5 million children ...
Obstructive lung disease is a category of respiratory disease characterized by airway obstruction.Many obstructive diseases of the lung result from narrowing (obstruction) of the smaller bronchi and larger bronchioles, often because of excessive contraction of the smooth muscle itself.
Bronchomalacia is a term for weak cartilage in the walls of the bronchial tubes, often occurring in children under a day. Bronchomalacia means 'floppiness' of some part of the bronchi. Patients present with noisy breathing and/or wheezing. There is collapse of a main stem bronchus on exhalation. If the trachea is also involved the term ...
Tracheobronchomegaly is a very rare congenital disorder of the lung primarily characterized by an abnormal widening of the upper airways. [2] The abnormally widened trachea and mainstem bronchi are associated with recurrent lower respiratory tract infection and copious purulent sputum production, eventually leading to bronchiectasis and other respiratory complications.
There is still much debate to whether pulmonary sequestration is a congenital problem or acquired through recurrent pulmonary infection. It is widely believed that extralobar pulmonary sequestrations are a result of prenatal pulmonary malformation while intralobar pulmonary sequestrations can develop due to recurrent pulmonary infections in adolescents and young adults.