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Non-plaque-induced gingival disease is an inflammation of the gingiva that does not result from dental plaque, but from other gingival diseases caused by bacterial, viral, fungal, or genetic sources. Although this gingival disease is less common than those which are plaque-induced, it can have a serious impact on the patient's overall health.
Dental plaque is a biofilm of microorganisms (mostly bacteria, but also fungi) that grows on surfaces within the mouth. It is a sticky colorless deposit at first, but when it forms tartar , it is often brown or pale yellow.
Periodontal pathogens are bacteria that have been shown to significantly contribute to periodontitis. Dental plaque, the precursor of periodontal disease, is a complex biofilm consisting mainly of bacteria, but also archaea, protozoa, fungi and viruses. Viruses that specifically infect bacteria—bacteriophages—are most common in the oral ...
Gum disease is the most common oral disease, with studies estimating that up to 80% of Americans have dealt with periodontal disease at some point during their lives. The prevalence of gum disease ...
Gum disease is typically caused by an accumulation of bacterial plaque — a sticky, colorless film — on the teeth and gums due to poor oral hygiene. ... Respiratory problems: The bacteria ...
Dental plaque biofilm-induced gingivitis; Non-dental-plaque-induced gingival disease; Dental plaque biofilm-induced gingivitis is often referred to as "localized inflammation initiated by microbial biofilm accumulation on teeth,". [2] Non-dental-plaque-induced gingival diseases are the most uncommon bacterial infection of the gingiva.
Periodontal disease, also known as gum disease, is a set of inflammatory conditions affecting the tissues surrounding the teeth. [5] In its early stage, called gingivitis, the gums become swollen and red and may bleed. [5] It is considered the main cause of tooth loss for adults worldwide.
The amount of plaque present is inconsistent with the amount and severity of tissue destruction [2] [33] but with a high plaque pathogenicity due to the presence of increased levels of bacteria like Aggregatibacter actinomycetemcomitans (A.a) and Porphyromonas Gingivalis (P.g). [2] Secondary features of LAP may also be present including; [2]
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