Search results
Results from the WOW.Com Content Network
Carditis (pl. carditides) is the inflammation of the heart. [1] It is usually studied and treated by specifying it as: [citation needed] Pericarditis is the ...
Laboratory values can show increased blood urea nitrogen , or increased blood creatinine in cases of uremic pericarditis. Generally, however, laboratory values are normal, but if there is a concurrent myocardial infarction (heart attack) or great stress to the heart, laboratory values may show increased cardiac markers like Troponin (I, T), CK ...
Myocarditis has been reported to be a major cause of sudden cardiac death (SCD) in infants, adolescents, and young adults, but the reported rates show wide variation (1 to 14 percent) among young people depending on differences in SCD definition and classification/ definition of myocarditis post-mortem as well as heterogeneity of study populations.
A complete blood count may show an elevated white count and a serum C-reactive protein may be elevated. Acute pericarditis is associated with a modest increase in serum creatine kinase MB (CK-MB). [ 5 ] and cardiac troponin I (cTnI), [ 6 ] [ 7 ] both of which are also markers for injury to the muscular layer of the heart.
Infective endocarditis is an infection of the inner surface of the heart (endocardium), usually the valves. [1] Signs and symptoms may include fever, small areas of bleeding into the skin, heart murmur, feeling tired, and low red blood cell count.
Late gadolinium enhancement can show enhancement of the pericardium due to fibroblast proliferation and neovascularization. [ 9 ] BNP blood test - tests for the existence of the cardiac hormone brain natriuretic peptide , which is only present in restrictive cardiomyopathy but not in constrictive pericarditis [ 12 ]
There are many causes of eosinophilia that may underlie eosinophilic myocarditis. These causes are classified as primary (i.e. a defect intrinsic to the eosinophil cell line), secondary (induced by an underlying disorder that stimulates the proliferation and activation of eosinophils), or idiopathic (i.e. unknown cause).
Management of carditis in acute rheumatic fever is controversial and based on dated literature. [55] Corticosteroids may be considered, especially in people with allergies to NSAIDs or severe disease, [ 48 ] although use of steroids may cause tissue atrophy, which could present challenges during future cardiac surgery for valve repair.