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Vertebrobasilar insufficiency (VBI) describes a temporary set of symptoms due to decreased blood flow in the posterior circulation of the brain. The posterior circulation supplies the medulla , pons , midbrain , cerebellum and (in 70-80% of people) supplies the posterior cerebellar artery to the thalamus and occipital cortex . [ 1 ]
Symptoms are repeated, brief periods of vertigo with movement, characterized by a spinning sensation upon changes in the position of the head. [1] This can occur with turning in bed or changing position. [3] Each episode of vertigo typically lasts less than one minute. [3] Nausea is commonly associated. [7] BPPV is one of the most common causes ...
vertebrobasilar insufficiency; optic neuritis; visual snow syndrome; Vitreous shrinkage or liquefaction, which is the most common cause of photopsia, causes a pull in vitreoretinal attachments, irritating the retina and causing it to discharge electrical impulses.
Brain ischemia has been linked to a variety of diseases or abnormalities. Individuals with sickle cell anemia, compressed blood vessels, ventricular tachycardia, plaque buildup in the arteries, blood clots, extremely low blood pressure as a result of heart attack, and congenital heart defects have a higher predisposition to brain ischemia in comparison to the average population.
TOS has been reported to cause stroke through theorized clot propagation towards the vertebral artery; [3] a similar mechanism could explain how TOS causes SSS. Presence of a cervical rib is a risk factor for both TOS and SSS. [citation needed] Takayasu's arteritis is a disease causing inflammation of arteries, including the subclavian artery ...
Other causes include Ménière's disease (12%), superior canal dehiscence syndrome, vestibular neuritis, vestibular paroxysmia, and visual vertigo. [16] [17] Any cause of inflammation such as common cold, influenza, and bacterial infections may cause transient vertigo if it involves the inner ear, as may chemical insults (e.g., aminoglycosides ...
A rare but severe complication of this disease is vertebrobasilar insufficiency. [7] This is a result of the vertebral artery becoming occluded as it passes up in the transverse foramen. The spinal joints become stiff in cervical spondylosis. Thus the chondrocytes which maintain the disc become deprived of nutrition and die.
The treatment for lateral pontine syndrome varies greatly, so there are different medications for different symptoms. [9] Sometimes blood thinning agents are prescribed to remove blood flow hindrance. [10] Other than these medications, physical therapy is also necessary [11]