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The bacteria Piscirickettsia salmonis causes the disease piscirickettsiosis, which has a mortality rate as high as 90% in certain salmonid fishes and is ubiquitous on Chilean salmon farms. Infected fish may or may not display external symptoms of infection, but they frequently display ulcers on the liver and kidney and often develop anemia.
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These symptoms negatively impact nutritional status, including decreased absorption of micronutrients, loss of appetite, weight loss, and intestinal blood loss that can often result in anemia. It may also cause physical and mental disabilities, delayed growth in children, and skin irritation around the anus and vulva.
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The fish responds by walling off the parasitic infection into a number of cysts that contain milky fluid. This fluid is an accumulation of a large number of parasites. Henneguya and other parasites in the myxosporean group have a complex lifecycle where the salmon is one of two hosts. The fish releases the spores after spawning.
Some fish show no external symptoms, but others show signs of infection that include bulging eyes, bloated abdomens, bruised-looking reddish tints to the eyes, skin, gills and fins. Some infected fish have open sores that may look like the lesions from other diseases or from lamprey attacks.
The symptoms the fish show are external and internal hemorrhaging, swelling of the vents and kidneys, boils, ulcers, liquefaction, and gastroenteritis. Furunculosis is commonly known as tail rot in fish and is common in goldfish and koi. Infected fish with open sores are able to spread the disease to other fish. [5]
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