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Aphakia Miosis caused by high doses of opiates . The person also shows ptosis of both eyelids and an inattentive look at the camera, a sign of altered level of consciousness caused by the sedative effect of the drug.
Aphakia is the absence of the lens of the eye, due to surgical removal, such as in cataract surgery, a perforating wound or ulcer, or congenital anomaly. It causes a loss of ability to maintain focus ( accommodation ), high degree of farsightedness ( hyperopia ), [ 1 ] and a deep anterior chamber .
Dilation and constriction of the pupil. Pupillary response is a physiological response that varies the size of the pupil, via the optic and oculomotor cranial nerve.. A constriction response (), [1] is the narrowing of the pupil, which may be caused by scleral buckles or drugs such as opiates/opioids or anti-hypertension medications.
Aphakia correction: High degree hypermetropia due to absence of lens (aphakia) is best corrected using intraocular lens implantation. [ citation needed ] Refractive lens exchange (RLE): A variation of cataract surgery where the natural crystalline lens is replaced with an artificial intraocular lens ; the difference is the existence of abnormal ...
When cycloplegic drugs are used as a mydriatic to dilate the pupil, the pupil in the normal eye regains its function when the drugs are metabolized or carried away. Some cycloplegic drugs can cause dilation of the pupil for several days.
Horner's syndrome, also known as oculosympathetic paresis, [1] is a combination of symptoms that arises when a group of nerves known as the sympathetic trunk is damaged. The signs and symptoms occur on the same side (ipsilateral) as it is a lesion of the sympathetic trunk.
A: Aphakia B: Rim artefact C: Chin slip D: Lens position E: Corneal opacity F: Keratoconus G: Ptosis H: Pupil Miosis - 1mm I: Pupil Miosis - 3mm Can be used for patients who are wheelchair users, hearing impaired, have postural and fixation problems and/or very low visual acuity [12] Provides a baseline measurement
Deletions or mutations to segments of mtDNA lead to defective function of oxidative phosphorylation. This may be made evident in highly oxidative tissues like skeletal muscle and heart tissue. However, extraocular muscles contain a volume of mitochondria that is several times greater than any other muscle group.