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Agonal breathing is a severe medical sign requiring immediate medical attention, as the condition generally progresses to complete apnea and preludes death. The duration of agonal respiration can range from two breaths to several hours of labored breathing.
A death rattle is noisy breathing that often occurs in someone near death. [1] Accumulation of fluids such as saliva and bronchial secretions in the throat and upper airways is the cause. [ 2 ] Those who are dying may lose their ability to swallow and may have increased production of bronchial secretions, resulting in such an accumulation. [ 3 ]
Respiratory arrest is a serious medical condition caused by apnea or respiratory dysfunction severe enough that it will not sustain the body (such as agonal breathing). Prolonged apnea refers to a patient who has stopped breathing for a long period of time. If the heart muscle contraction is intact, the condition is known as respiratory arrest.
Signs of death or strong indications that a human is no longer alive are: Respiratory arrest (no breathing); Cardiac arrest (no pulse); Brain death (no neuronal activity); The heart and lungs are vital organs for human life due to their ability to properly oxygenate human blood (lungs) and distribute this blood to all vital organs (heart).
Hyperpnea – fast and deep breathing; Hyperventilation – increased breathing that causes CO 2 loss; Hypopnea – slow and shallow breathing; Hypoventilation – decreased breathing that causes CO 2 gain; Labored breathing – physical presentation of respiratory distress; Tachypnea – increased breathing rate
Clinical death is the medical term for cessation of blood circulation and breathing, the two criteria necessary to sustain the lives of human beings and of many other organisms. [1] It occurs when the heart stops beating in a regular rhythm, a condition called cardiac arrest .
Various breathing abnormalities . Ataxic respirations are one of many unique respiration styles in an ill patient. There is an apparent controversy surrounding the novelty of ataxic respirations versus the well-known Cheyne-Stokes and cluster respirations, which Dr. Camille Biot deemed mutually exclusive. [3]
Causes may include heart failure, kidney failure, narcotic poisoning, intracranial pressure, and hypoperfusion of the brain (particularly of the respiratory center). The pathophysiology of Cheyne–Stokes breathing can be summarized as apnea leading to increased CO 2 which causes excessive compensatory hyperventilation, in turn causing decreased CO 2 which causes apnea, restarting the cycle.