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It was created in Lima by the Congregation of Religious of the Sacred Heart (present in education in Peru since 1876) and authorized by Supreme Decree 71 of December 24, 1962, given under the government of the Military junta presided over by the general Ricardo Pérez Godoy. Its peculiarity was that it was designed to be only for women.
The Church of the Immaculate Heart of Mary (Spanish: Iglesia del Inmaculado Corazón de María), commonly known as The Dome (Spanish: La Cúpula), [1] is a Catholic church located in the intersection of Antonio José de Sucre Avenue and Jirón 28 de Julio, in Magdalena del Mar District, Lima, Peru.
The Sacred Heart Church (Spanish: Iglesia del Sagrado Corazón de Jesús), also known as the Church of the Orphans (Spanish: Iglesia de los Huérfanos), is a Catholic church dedicated to the Sacred Heart, located at the intersection of Jirón Azángaro and Jirón Apurímac in the historic centre of Lima, Peru.
The main one (the one in the center) is called Puerta del Perdón, the right side is called Puerta de la Epístola and the left side is called Puerta del Evangelio. There are also two side doors, one that leads to the Calle de Judíos (right side) and another that leads to the Patio de los Naranjos (inner courtyard attached to the cathedral).
The Central Reserve Bank of Peru (Spanish: Banco Central de Reserva del Perú; BCRP) is the Peruvian central bank.It mints and issues metal and paper money, the sol.. Its branch in Arequipa was established in 1871, [citation needed] and it served the city by issuing money as well as maintaining a good reputation for savings accounts in Southern Peru.
The Plaza Mayor (English: Town square), also known as the Plaza de Armas, [1] is the main public square of the historic centre of the city of Lima, Peru.It dates back to the foundation of the city by Francisco Pizarro on January 18, 1535.
The symbol BCR is derived from breakpoint cluster region, a gene which encodes a protein that acts as a guanine nucleotide exchange factor for Rho GTPase proteins. [7] Translocation results in an oncogenic BCR-ABL1 gene fusion that can be found on the shorter derivative chromosome 22. This gene encodes for a BCR-ABL1 fusion protein.
Nilotinib is a selective Bcr-Abl kinase inhibitor. [13] [18] Nilotinib is 10-30 fold more potent than imatinib in inhibiting activity of the Bcr-Abl tyrosine kinase and proliferation of Bcr-Abl expressing cells. [12] [18] [20] [21] The drug effectively inhibits the auto phosphorylation of Bcr-Abl on Tyr-177 that is involved in CML pathogenesis ...