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  2. Asthma - Wikipedia

    en.wikipedia.org/wiki/Asthma

    Asthma is a common long-term inflammatory disease of the airways of the lungs. [4] Asthma occurs when allergens, pollen, dust, or other particles, are inhaled into the lungs, causing the bronchioles to constrict and produce mucus, which then restricts oxygen flow to the alveoli.

  3. Pathophysiology of asthma - Wikipedia

    en.wikipedia.org/wiki/Pathophysiology_of_asthma

    During an asthma episode, inflamed airways react to environmental triggers such as smoke, dust, or pollen. The airways narrow and produce excess mucus, making it difficult to breathe. In essence, asthma is the result of an immune response in the bronchial airways. [3]

  4. Phlegm - Wikipedia

    en.wikipedia.org/wiki/Phlegm

    Mucous membranes produce histamines and make more phlegm. Red: Indicates dry air. A nasal spray can be used to alleviate symptoms of a dry nose and throat. It can also occur due to blood (such as if the person had or has a bleeding nose, or a lung malignancy).

  5. Got snot? Here's what your mucus tells you about allergies ...

    www.aol.com/lifestyle/got-snot-heres-mucus-tells...

    "Mucus is very helpful in terms of our immune system," Elliott says. "When you have a foreign particle like pollen or a virus or bacteria, your mucosal cells — which produce mucus — are ...

  6. Yep, Allergies Might Be to Blame for Your Upset Stomach ... - AOL

    www.aol.com/news/fall-allergies-might-blame...

    “Different allergens and pollen spike throughout the year, and it can sometimes be difficult to diagnose gastrointestinal issues due to seasonal allergies because oftentimes upper respiratory ...

  7. Airway remodelling - Wikipedia

    en.wikipedia.org/wiki/Airway_remodelling

    Airway remodelling is a multifaceted process involving multiple airway tissues. These include goblet cell hyperplasia, leading to increased mucus production, and airway smooth muscle hypertrophy (or smooth muscle cell hyperplasia), leading to the release of pro-inflammatory and pro-fibrotic messengers contributing to subepithelial fibrosis.

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