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Local signs included inflammation of the gums and Gingival reddening (Hyperemia) most commonly presenting in posterior teeth. A study shows that 70.5% of children between 0–36 months showed signs and symptoms of tooth eruption whether it being a fever, Gingival irrational and/or drooling.
Gingivitis is a non-destructive disease that causes inflammation of the gums; [1] ulitis is an alternative term. [2] The most common form of gingivitis, and the most common form of periodontal disease overall, is in response to bacterial biofilms (also called plaque) that are attached to tooth surfaces, termed plaque-induced gingivitis.
As gingivitis progresses further and is not treated, it may progress into periodontitis. Periodontal disease is when the gums surrounding the teeth become swollen causing surrounding plaque to build up. If left untreated can cause the teeth to become loose due to weak gums. Periodontal disease can compromise factors such as: [4] Gingiva
“Introduce a half-stick first and remind your child that this is not food or candy, but something to chew and spit out later,” advises Dr. Denise Scott, a pediatrician and expert with JustAnswer.
Early stages of decay (white spot lesions) and initial enamel damage are managed with non-invasive preventive methods and are closely monitored over time. High-risk children typically need more intensive treatment. This may include early restorative work to repair and address any existing decay to prevent further deterioration of the teeth.
One of the earliest signs in the formation of a tooth that can be seen microscopically is the distinction between the vestibular lamina and the dental lamina. It occurs in the sixth to seventh week of the embryonic life. The dental lamina connects the developing tooth bud to the epithelial layer of the mouth for a significant time. [21]
Herpetic gingivostomatitis is an infection caused by the herpes simplex virus (HSV). The HSV is a double-stranded DNA virus categorised into two types; HSV-1 and HSV-2.HSV-1 is predominantly responsible for oral, facial and ocular infections whereas HSV-2 is responsible for most genital and cutaneous lower herpetic lesions.
While in some individuals gingivitis never progresses to periodontitis, [14] periodontitis is always preceded by gingivitis. [15] In 1976, Page & Schroeder [16] introduced an innovative new analysis of periodontal disease based on histopathologic and ultrastructural features of the diseased gingival tissue.
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