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In fluid dynamics, a moving shock is a shock wave that is travelling through a fluid (often gaseous) medium with a velocity relative to the velocity of the fluid already making up the medium. [1] As such, the normal shock relations require modification to calculate the properties before and after the moving shock.
Shock is the state of insufficient blood flow to the tissues of the body as a result of problems with the circulatory system. Initial symptoms of shock may include weakness, tachycardia, hyperventilation, sweating, anxiety, and increased thirst. [1] This may be followed by confusion, unconsciousness, or cardiac arrest, as complications worsen. [1]
Second, medical roots generally go together according to language, i.e., Greek prefixes occur with Greek suffixes and Latin prefixes with Latin suffixes. Although international scientific vocabulary is not stringent about segregating combining forms of different languages, it is advisable when coining new words not to mix different lingual roots.
Vital signs in obstructive shock may show hypotension, tachycardia, and/or hypoxia. A physical exam include be thorough, including jugular vein exam, cardiac and lung exams, and assessing skin tone and temperature. [22] Response to fluids may aid in diagnosis. [3] Labs including a metabolic panel can assess electrolytes and kidney and liver ...
Vasodilatory shock, vasogenic shock, or vasoplegic shock is a medical emergency belonging to shock along with cardiogenic shock, septic shock, allergen-induced shock and hypovolemic shock. When the blood vessels suddenly relax, it results in vasodilation .
shock resuscitation unit ss ss: one-half (ss either with or without a bar over them) (from Latin semis) SS: hemoglobin SS (HbSS) (see in sickle-cell disease = SS disease) subserosal Sjogren's Syndrome S/S S/Sx: signs and symptoms: SSC: secondary sex characteristics: SSE: sterile speculum exam SSEP: somatosensory evoked potential: SSI: sliding ...
Neurogenic shock results from damage to the spinal cord above the level of the 6th thoracic vertebra. [5] It is found in about half of people who have a spinal cord injury within the first 24 hours, and usually persists for one to three weeks. [5] Neurogenic shock may be caused by severe brain injury. [6]
HMGB1 is a major mediator of endotoxin shock [19] and is recognized as a DAMP by certain immune cells, triggering an inflammatory response. [12] It is known to induce inflammation by activating NF-κB pathway by binding to TLR, TLR4, TLR9, and RAGE (receptor for advanced glycation end products). [ 20 ]