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Pulpitis is inflammation of dental pulp tissue. The pulp contains the blood vessels, the nerves, and connective tissue inside a tooth and provides the tooth's blood and nutrients. Pulpitis is mainly caused by bacterial infection which itself is a secondary development of caries (tooth decay). It manifests itself in the form of a toothache. [1]
Pulpitis is reversible when the pain is mild to moderate and lasts for a short time after a stimulus (for instance cold); or irreversible when the pain is severe, spontaneous, and lasts a long time after a stimulus. Left untreated, pulpitis may become irreversible, then progress to pulp necrosis (death of the pulp) and apical periodontitis ...
Pulp stones generally do not have significant clinical implications as they are usually not a source of pain, discomfort or any form of pulpitis. [5] [12] However, when the tooth concerned will undergo endodontic treatment such as root canal treatment, presence of large pulp stones will be clinically significant. [12] [13]
Dental pulpal testing is a clinical and diagnostic aid used in dentistry to help establish the health of the dental pulp within the pulp chamber and root canals of a tooth. . Such investigations are important in aiding dentists in devising a treatment plan for the tooth being test
Periapical periodontitis of some form is a very common condition. The prevalence of periapical periodontitis is generally reported to vary according to age group, e.g. 33% in those aged 20–30, 40% in 30- to 40-year-olds, 48% in 40- to 50-year-olds, 57% in 50- to 60-year-olds and 62% in those over the age of 60. [13]
An x-ray of a root canal operation. Before endodontic therapy is carried out, a correct diagnosis of the dental pulp and the surrounding periapical tissues is required. This allows the endodontist to choose the most appropriate treatment option, allowing preservation and longevity of the tooth and surrounding tissues.
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Ongoing tooth infections, like pulpitis, release chemicals that attract immune cells and activate osteoblasts. Osteoblasts create extra bone in response to inflammation, making the area look more sclerotic. Unlike typical infections, condensing osteitis doesn't destroy bone or produce pus it just adds more bone to the affected area. [4] [6]