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Pathophysiology of obesity is the study of disordered physiological processes that cause, result from, or are otherwise associated with obesity. A number of possible pathophysiological mechanisms have been identified which may contribute in the development and maintenance of obesity.
The pathogenesis of sarcopenic obesity involves multiple factors, including aging, lack of physical activity, malnutrition/vitamin imbalances, insulin resistance, and hormonal changes -> body composition changes. The exact pathophysiology is not well understood, however these factors have been studied in the production of sarcopenic obesity.
Obesity is a major cause of disability and is correlated with various diseases and conditions, particularly cardiovascular diseases, type 2 diabetes, obstructive sleep apnea, certain types of cancer, and osteoarthritis. [2] [12] [13] Obesity has individual, socioeconomic, and environmental causes.
The pathophysiology of obesity involves many possible pathophysiological mechanisms involved in its development and maintenance. [25] [26] This field of research had been almost unapproached until the leptin gene was discovered in 1994 by J. M. Friedman's laboratory. [27] These investigators postulated that leptin was a satiety factor.
Death rate from obesity, 2019. Obesity is a risk factor for many chronic physical and mental illnesses.. The health effects of being overweight but not obese are controversial, with some studies showing that the mortality rate for individuals who are classified as overweight (BMI 25.0 to 29.9) may actually be lower than for those with an ideal weight (BMI 18.5 to 24.9). [1]
Like many other medical conditions, obesity is the result of an interplay between environmental and genetic factors. [2] [3] Studies have identified variants in several genes that may contribute to weight gain and body fat distribution; although, only in a few cases are genes the primary cause of obesity.
Metabolic basis of obesity (Springer) Obesity epidemiology, pathogenesis, and treatment: a multidisciplinary approach (Apple/CRC) Childhood obesity: prevalence, pathophysiology, and management (Apple/CRC) The Year in Diabetes and Obesity (Annals of the New York Academy of Sciences)_review series; Highly Cited Articles (more than 1300 citations)
An association between viruses and obesity has been found in humans, as well as a number of different animal species. The amount that these associations may have contributed to the rising rate of obesity is yet to be determined. [5] A fat virus is the popular name for the notion that some forms of obesity in humans and animals have a viral source.