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There are several causes of toxic optic neuropathy. [1] Among these are: ingestion of methanol (wood alcohol), ethylene glycol (automotive antifreeze), disulfiram (used to treat chronic alcoholism), halogenated hydroquinolones (amebicidal medications), ethambutol and isoniazid (tuberculosis treatment), and antibiotics such as linezolid and chloramphenicol as well as chloroquine and the related ...
Major symptoms are sudden loss of vision (partial or complete), sudden blurred or "foggy" vision, and; pain on movement of the affected eye. [4] [5] [2]Many patients with optic neuritis may lose some of their color vision in the affected eye (especially red), with colors appearing subtly washed out compared to the other eye.
Conjunctivitis, also known as pink eye or Madras eye, [4] [5] is inflammation of the conjunctiva and the inner surface of the eyelid. [6] It makes the eye appear pink or reddish. [1] Pain, burning, scratchiness, or itchiness may occur. [1] The affected eye may have increased tears or be "stuck shut" in the morning. [1] Swelling of the sclera ...
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Kirsty Drury thought her bloodshot eye was conjunctivitis - it turned out to be symptomatic of a brain tumour. Skip to main content. 24/7 Help. For premium support please call: 800-290-4726 ...
Watery eyes – due to excessive tearing. [7] Red eyes – due to dilated blood vessels on the sclera. [7] Swollen eyelids – due to inflammation. [7] Crusting at the eyelid margins/base of the eyelashes/medial canthus, generally worse on waking – due to excessive bacterial buildup along the lid margins. [4] [5] [7]
It usually manifests in 1–3 hours after the first dose of antibiotics as fever, chills, rigor, hypotension, headache, tachycardia, hyperventilation, vasodilation with flushing, myalgia (muscle pain), exacerbation of skin lesions and anxiety. The intensity of the reaction indicates the severity of inflammation.
Ocular myasthenia gravis (MG) is a disease of the neuromuscular junction resulting in hallmark variability in muscle weakness and fatigability. MG is an autoimmune disease where anomalous antibodies are produced against the naturally occurring acetylcholine receptors in voluntary muscles.