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The most common cause of polyuria in both adults and children is uncontrolled diabetes mellitus, [6] which causes osmotic diuresis; when glucose levels are so high that glucose is excreted in the urine. Water follows the glucose concentration passively, leading to abnormally high urine output. [citation needed]
Excessive urination that’s not due to copious water or beverage consumption can have multiple causes, including overactive bladder syndrome, diabetes, a urinary tract infection or medications ...
The most common cause of urinary frequency in older men is an enlarged prostate. [2] Frequent urination is strongly associated with frequent incidents of urinary urgency, which is the sudden need to urinate. It is often, though not necessarily, associated with urinary incontinence and polyuria (large total volume of urine). However, in other ...
Diuresis (/ ˌ d aɪ j ʊ ˈ r iː s ɪ s /) is the excretion of urine, especially when excessive ().The term collectively denotes the physiologic processes underpinning increased urine production by the kidneys during maintenance of fluid balance.
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Many diuretic drugs take advantage of this mechanism to treat medical conditions like hypernatremia and hypertension, which involve excess blood volume. Excess natriuresis can be caused by: Medullary cystic disease; Bartter syndrome; Diuretic phase of acute tubular necrosis; Some diuretics; Primary renal diseases; Congenital adrenal hyperplasia
Polyuria (excessive urine production) of which, in turn, the most frequent causes are: uncontrolled diabetes mellitus, primary polydipsia (excessive fluid drinking), central diabetes insipidus and nephrogenic diabetes insipidus. [19] Polyuria generally causes urinary urgency and frequency, but does not necessarily lead to incontinence.
Excessive ADH causes an inappropriate increase in the reabsorption in the kidneys of solute-free water ("free water"): excess water moves from the distal convoluted tubules (DCTs) and collecting tubules of the nephrons – via activation of aquaporins, the site of the ADH receptors – back into the circulation. This has two consequences.
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