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Bronchiectasis is a disease in which there is permanent enlargement of parts of the airways of the lung. [5] Symptoms typically include a chronic cough with mucus production. [ 3 ] Other symptoms include shortness of breath , coughing up blood , and chest pain. [ 2 ]
Culture results at first were negative. The source of the outbreak was traced to the air-conditioning cooling towers on the nursing home's roof. [22] 2005: Fredrikstad, Norway: Factory: Air scrubber: 103: 10: 9.7%: At least 103 people became ill and ten died from Legionnaires' disease caused by bacteria growing in an air scrubber of a nearby ...
Obstructive lung disease is a category of respiratory disease characterized by airway obstruction.Many obstructive diseases of the lung result from narrowing (obstruction) of the smaller bronchi and larger bronchioles, often because of excessive contraction of the smooth muscle itself.
Areas of emphysema are abnormal, air-filled spaces that usually do not have visible walls, [5] and bullae are very thin walled (<1 mm). [2] Cystic bronchiectasis is irreversible bronchial dilation, which is permanent widening of the bronchioles (small airways) in the lung. [2]
Pulmonary edema (British English: oedema), also known as pulmonary congestion, is excessive fluid accumulation in the tissue or air spaces (usually alveoli) of the lungs. [1] This leads to impaired gas exchange , most often leading to shortness of breath ( dyspnea ) which can progress to hypoxemia and respiratory failure .
Respiratory virus season is officially here in the U.S., making it a prime time to catch a cold. And because the average adult gets two or three colds a year, you could be dealing with an ...
Bronchorrhea is the production of more than 100 mL per day of watery sputum. [1] Chronic bronchitis is a common cause, but it may also be caused by asthma, [2] pulmonary contusion, [3] bronchiectasis, tuberculosis, cancer, scorpion stings, severe hypothermia and poisoning by organophosphates and other poisons.
In 1976, the first report of the occurrence of familial bronchiectasis in siblings was published, and it supported the theory that WCS was congenital, based on the uniformity of the cartilaginous defect. [13] [9] [10] It may have been the result of an autosomal recessive mutation, but the specific gene has not yet been identified. [3]