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Leukoplakia is, therefore, the most common premalignant lesion that occurs in the mouth. [41] Leukoplakia is more common in middle-aged and elderly males. [30] The prevalence increases with increasing age. [2] In areas of the world where smokeless tobacco use is common, there is a higher prevalence. [2]
Hairy leukoplakia is a white patch on the side of the tongue with a corrugated or hairy appearance. It is caused by Epstein-Barr virus (EBV) and occurs usually in persons who are immunocompromised , especially those with human immunodeficiency virus infection/ acquired immunodeficiency syndrome (HIV/AIDS).
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The tongue is prone to several pathologies including glossitis and other inflammations such as geographic tongue, and median rhomboid glossitis; burning mouth syndrome, oral hairy leukoplakia, oral candidiasis (thrush), black hairy tongue, bifid tongue (due to failure in fusion of two lingual swellings of first pharyngeal arch) and fissured tongue.
Proliferative verrucous leukoplakia; Pyogenic granuloma (eruptive hemangioma, granulation tissue-type hemangioma, granuloma gravidarum, lobular capillary hemangioma, pregnancy tumor, tumor of pregnancy) Pyogenic granuloma; Pyostomatitis vegetans; Recurrent aphthous stomatitis (aphthosis, canker sores, recurrent oral aphthae)
Oral cancer, also known as oral cavity cancer, tongue cancer or mouth cancer, is a cancer of the lining of the lips, mouth, or upper throat. [6] In the mouth, it most commonly starts as a painless red or white patch, that thickens, gets ulcerated and continues to grow.
Bowen's disease presents as leukoplakia on the shaft. Around a third of cases progress to SCC. Erythroplasia of Queyrat, a variation of Bowen's disease, presents as erythroplakia on the glans. Bowenoid papulosis, which histologically resembles Bowen disease, presents as reddish papules. [9]
Diagnosis is mainly clinical, based on the history and clinical appearance. The differential diagnosis includes other oral white lesions such as Leukoplakia, squamous cell carcinoma, oral candidiasis, lichen planus, white sponge nevus and contact stomatitis. [7] In contrast to pseudomembraneous candidiasis, this white patch cannot be wiped off. [7]