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Dizziness Are some people more likely to have motion sickness than others? While researchers don’t know why, Qing says women are more prone to motion sickness, as are children between ages 2 and 9.
Scopolamine, also known as hyoscine, [9] or Devil's Breath, [10] is a natural or synthetically produced tropane alkaloid and anticholinergic drug that is used as a medication to treat motion sickness [11] and postoperative nausea and vomiting.
Hyoscine butylbromide, also known as scopolamine butylbromide [4] and sold under the brandname Buscopan among others, [5] is an anticholinergic medication used to treat abdominal pain, esophageal spasms, bladder spasms, biliary colic, [6] and renal colic.
Benign paroxysmal positional vertigo, vestibular migraine, stroke [2] Prevention: Avoidance of triggers [2] Treatment: Behavioral measures, medications [3] Medication: Scopolamine, dimenhydrinate, dexamphetamine [3] Prognosis: Generally resolve within a day [2] Frequency: Nearly all people with sufficient motion; roughly one-third highly ...
The first time you experience vertigo, it can be an unsettling -- even scary -- experience. A slight shift of your head and you feel as if you're wildly spinning, or the world is spinning around you.
Medical treatment with anti-vertigo medications may be considered in acute, severe exacerbation of BPPV, but in most cases are not indicated. These primarily include drugs of the antihistamine and anticholinergic class, such as meclizine [9] and hyoscine butylbromide (scopolamine), respectively. The medical management of vestibular syndromes ...
Treatment may include drinking plenty of water or other fluids (unless the lightheadedness is the result of water intoxication in which case drinking water is quite dangerous). If a patient is unable to keep fluids down from nausea or vomiting, they may need intravenous fluids such as Ringer's lactate solution .
Methscopolamine, a methylated derivative of scopolamine, is a muscarinic antagonist structurally similar to the neurotransmitter acetylcholine. Its mechanism of action involves blocking the muscarinic acetylcholine receptors. It was patented in 1902 and approved for medical use in 1947. [3]