Search results
Results from the WOW.Com Content Network
A review for Boston Magazine called Sinclair "one of science's most controversial figures" and said many in the scientific community were skeptical of claims he made about human longevity. University of Alabama biology professor Steven N. Austad said, "David is a good friend, but I do think he's been guilty of making excessive claims."
David Andrew Sinclair AO (born June 26, 1969) is an Australian-American biologist and academic known for his research on aging and epigenetics. Sinclair is a professor of genetics at Harvard Medical School and the founding director of the Paul F. Glenn Laboratories for the Biological Mechanisms of Aging at Harvard.
Guarente's rivalry with Sinclair, which began in 2002 when Sinclair challenged Guarante's description of how sir2 might be involved in aging at a scientific meeting at Cold Spring Harbor Laboratory, was the subject of an article in Science in 2004. [20] In 2014 Guarente co-founded the dietary supplement company Elysium Health. [13] [21]
The global lifespan is nearly a decade longer than the average health span. The study found that an average global citizen lives 9.6 fewer healthy years than they live altogether — so, for ...
[42] [43] [44] After writing a favorable review of Steven Austad's book Methuselah's Zoo, [45] he reviewed Lifespan: Why We Age – and Why We Don't Have To by David A. Sinclair, summarizing it as "an influential source of misinformation on longevity, featuring counterfactual claims about longevity genes being conserved between yeast and humans ...
“Every additional step you take is an investment in your brain health,” Lydia Bazzano, MD, PhD, director of the Tulane Center for Lifespan Epidemiology Research at Tulane University, told ...
Savannah Rep's most recent production, 'The Lifespan of a Fact,' based upon the real-life struggle between an earnest fact-checker and celebrated essayist, confronts the nature of truth.
In 1993, Kenyon's discovery that a single-gene mutation could double the lifespan of C. elegans and that this could be reversed by a second mutation in daf-16m, [4] sparked an intensive study of the molecular biology of aging, including work by Leonard Guarente and David Sinclair. [1]