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Since stroke is an essential part of vascular dementia, [13] the goal is to prevent new strokes. This is attempted through reduction of stroke risk factors, such as high blood pressure , high blood lipid levels , atrial fibrillation , or diabetes mellitus .
Anything that affects brain function (including stroke or other conditions that compromise blood flow) can cause cognitive issues and even dementia. Vascular dementia can happen after a stroke ...
Brain scans may show evidence of multiple strokes of different sizes in various locations. People with vascular dementia tend to have risk factors for disease of the blood vessels, such as tobacco use, high blood pressure, atrial fibrillation, high cholesterol, diabetes, or other signs of vascular disease such as a previous heart attack or angina.
Cerebral atherosclerosis is a type of atherosclerosis where build-up of plaque in the blood vessels of the brain occurs. Some of the main components of the plaques are connective tissue, extracellular matrix, including collagen, proteoglycans, fibronectin, and elastic fibers; crystalline cholesterol, cholesteryl esters, and phospholipids; cells such as monocyte derived macrophages, T ...
“A stroke occurs when the central nervous system is injured due to a vascular cause, so that could be due to a blockage, which accounts for about 80% of strokes in the United States, or a ...
Having a higher biological than chronological age seemingly led to an elevated risk of dementia, especially vascular; ischemic stroke, from a blood clot in the brain; and ALS, a neurodegenerative ...
Another symptom which should be monitored is Type 2 diabetes, which can lead to vascular dementia. [14] Also linked with vascular dementia issues is high cholesterol; furthermore, this risk factor is related to cognitive decline, stroke, and Alzheimer's disease. [14] It is estimated that within 20 years, worldwide prevalence will increase twofold.
Binswanger's disease is a type of subcortical vascular dementia caused by white matter atrophy to the brain. However, white matter atrophy alone is not sufficient for this disease; evidence of subcortical dementia is also necessary.