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Dicamba is toxic to conifer species but is in general less toxic to grasses. [2] Dicamba is a synthetic auxin that functions by increasing plant growth rate, leading to senescence and cell death. [2] [11] The growth regulating properties of dicamba were first discovered by Zimmerman and Hitchcock in 1942. [12]
In vivo dermal sensitization studies according to OECD 429 [3] confirmed DIC is a strong skin sensitizer, showing a response at 0.20 wt% in the Local Lymph Node Assay (LLNA) placing it in Globally Harmonized System of Classification and Labelling of Chemicals (GHS) Dermal Sensitization Category 1A. [4]
The other auxin now used in comparable amounts to 2,4-D is dicamba, where the 2019 figure was 30,000,000 pounds (14,000,000 kg). [7] It is a benzoic acid rather than a phenoxyacetic acid whose use has grown rapidly since 2016 as crops genetically modified to be resistant to it have been cultivated. [8]
Approval has been granted to grow crops engineered to be resistant to the herbicides 2,4-dichlorophenoxyacetic acid, [8] dicamba, [9] glufosinate [10] glyphosate, [11] sulfonylurea, [12] oxynil [13] mesotrione [14] and isoxaflutole [15] Most herbicide resistant GM crops have been engineered for glyphosate tolerance, in the USA 93% of soybeans ...
The international trade of 2,4,5-T is restricted by the Rotterdam Convention. 2,4,5-T has since largely been replaced by dicamba and triclopyr. Human health effects from 2,4,5-T at low environmental doses or at biomonitored levels from low environmental exposures are unknown.
Chemical structure of Dicamba, 3,6-dichloro-2-methoxybenzoic acid. Dicamba drift is a particular problem, as has been recognized since at least 1979. [20] The effects have been noted for many crops: grapes, tomatoes, soybeans. [21] [22] In 2017, Dicamba-resistant soybeans and cotton were approved for use in the US. This new technology worsened ...
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