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Transient lingual papillitis is generally diagnosed based on patient presentation, meaning where it is located in the mouth and how big the bump is. [8] The visual presentation can also accompany various signs and symptoms such as difficulty eating, having a "strawberry tongue", increased saliva production, and a burning or tingling sensation. [9]
The exact appearance of the lesion is variable. Leukoplakia may be white, whitish yellow or grey. [29] The size can range from a small area to much larger lesions. [29] The most common sites affected are the buccal mucosa, the labial mucosa and the alveolar mucosa, [30] although any mucosal surface in the mouth may be involved. [2]
The lesion is a white patch, which almost exclusively occurs on the lateral surfaces of the tongue, although rarely it may occur on the buccal mucosa, soft palate, pharynx or esophagus. [5] The lesion may grow to involve the dorsal surface of the tongue. The texture is vertically corrugated ("hairy") or thickly furrowed and shaggy in appearance.
Tongue lesions are very common. For example, in the United States one estimated point prevalence was 15.5% in adults. [10] Tongue lesions are more common in persons who wear dentures and tobacco users. [10] The most common tongue conditions are geographic tongue, followed by fissured tongue and hairy tongue. [10]
Iron-deficiency anemia is mainly caused by blood loss, such as may occur during menses or gastrointestinal hemorrhage.This often results in a depapilled, atrophic glossitis, giving the tongue a bald and shiny appearance, along with pallor (paleness) of the lips and other mucous membranes a tendency towards recurrent oral ulceration, [6] and cheilosis (swelling of the lips). [7]
Squamous cell papilloma of the mouth or throat is generally diagnosed in people between the ages of 30 and 50, [1] and is normally found on the inside of the cheek, on the tongue, or inside of lips. Oral papillomas are usually painless, and not treated unless they interfere with eating or are causing pain. [ 1 ]
Leukoedema lesions disappear when the mucosa is stretched, which helps to differentiate it from other white lesions in the mouth. [2] The differential diagnosis is with leukoplakia , oral candidiasis , oral lichen planus , white sponge nevus , morsicatio buccarum , [ 3 ] hereditary benign intraepithelial dyskeratosis and dyskeratosis congenita.
The lesions are harmless; no treatment is indicated beyond reassurance unless the person requests it. The most common and simple treatment is the construction of a specially made acrylic prosthesis that covers the biting surfaces of the teeth and protects the cheek, tongue, and labial mucosa (an occlusal splint). This is either employed in the ...
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