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No loss of body function occurs, and the effects of alopecia areata are psychological (loss of self-image due to hair loss), although these can be severe. Loss of hair also means the scalp burns more easily in the sun. Patients may also have aberrant nail formation because keratin forms both hair and nails. [citation needed]
Many treatments have been explored, including immunomodulatory agents such as imiquimod. [4] Tofacitinib citrate may also have benefits. In June 2014, a 25-year-old man with almost no hair on his body was reported to have grown a full head of hair, as well as eyebrows, eyelashes, and facial, armpit, and other hair, following eight months of treatment. [5]
The patient can have thinning and loss of hair around the whole scalp, at the occipital scalp or around the scalp in odd, irregular patches. [ 7 ] [ 13 ] Loose anagen syndrome hairs at the occipital scalp can be knotted easily when there's movement of those hairs against the pillow at night.
Symptoms of Hair Loss. Classically, symptoms of hair loss in men include a receding hairline (particularly over the temples, creating a characteristic “M” shape) and signs of balding at the ...
Risk Factors for Hair Loss in Women. There are quite a few risk factors for hair loss in women. They include: Age. FPHL usually develops around midlife and continues to progress as you get older.
The cause of male-pattern hair loss is a combination of genetics and male hormones; the cause of female pattern hair loss is unclear; the cause of alopecia areata is autoimmune; and the cause of telogen effluvium is typically a physically or psychologically stressful event. [4] Telogen effluvium is very common following pregnancy. [4]
It can cause diffuse hair loss across the entire scalp, a receding hairline, a bald patch, or a mix of hair loss symptoms. Male pattern baldness (androgenetic alopecia) is a common issue that can ...
Alopecia totalis is the loss of all hair on the head and face.Its causes are unclear, but believed to be autoimmune.Research suggests there may be a genetic component linked to developing alopecia totalis; the presence of DRB1*0401 and DQB1*0301, both of which are human leukocyte antigens (HLA), were found to be associated with long-standing alopecia totalis.
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