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Hyperventilation syndrome (HVS), also known as chronic hyperventilation syndrome (CHVS), dysfunctional breathing hyperventilation syndrome, cryptotetany, [1] [2] spasmophilia, [3] [4] [5] latent tetany, [4] [5] and central neuronal hyper excitability syndrome (NHS), [3] is a respiratory disorder, psychologically or physiologically based, involving breathing too deeply or too rapidly ...
Hyperventilation is irregular breathing that occurs when the rate or tidal volume of breathing eliminates more carbon dioxide than the body can produce. [ 1 ] [ 2 ] [ 3 ] This leads to hypocapnia , a reduced concentration of carbon dioxide dissolved in the blood.
The hyperventilation leads to an excessive elimination of carbon dioxide (CO 2) whereas no significant additional amounts of oxygen can be stocked in the body. As only carbon dioxide is responsible for the breathing stimulus, [ citation needed ] after hyperventilation, breath can be held longer until cerebral hypoxia occurs.
When this happens, part of the heart muscle can stop functioning or even die. This event is called a myocardial infarction or heart attack, and can lead to permanent heart damage or be fatal ...
Self-induced hypocapnia through hyperventilation is the basis for the dangerous schoolyard fainting game. [ citation needed ] Deliberate hyperventilation has been used by underwater breath-hold divers for the purpose of extending dive time as it effectively reduces respiratory drive due to low CO 2 levels.
Prolonged apnea refers to a patient who has stopped breathing for a long period of time. If the heart muscle contraction is intact, the condition is known as respiratory arrest. An abrupt stop of pulmonary gas exchange lasting for more than five minutes may permanently damage vital organs, especially the brain.
While heart disease and stroke deaths had been decreasing in both rural and urban communities before 2019, they shot up with the arrival of the pandemic in 2020, reversing decades of progress.
Causes may include heart failure, kidney failure, narcotic poisoning, intracranial pressure, and hypoperfusion of the brain (particularly of the respiratory center). The pathophysiology of Cheyne–Stokes breathing can be summarized as apnea leading to increased CO 2 which causes excessive compensatory hyperventilation, in turn causing decreased CO 2 which causes apnea, restarting the cycle.
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