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A hyperintensity or T2 hyperintensity is an area of high intensity on types of magnetic resonance imaging ... presence of severe WMH and medial temporal lobe atrophy ...
The temporal lobe is located beneath the lateral fissure on both cerebral hemispheres of the mammalian brain. [3] The temporal lobe is involved in processing sensory input into derived meanings for the appropriate retention of visual memory, language comprehension, and emotion association. [4]: 21 Temporal refers to the head's temples.
The temporal lobe is involved in processing sensory input into derived meanings for the appropriate retention of visual memories, language comprehension, and emotion association. [10]: 21 Within the temporal lobe is an area of the brain called the hippocampus which is associated with forming new memories and learning new things. The hippocampus ...
Brain MRI is the mainstay of initial investigation pointing to limbic lobe pathology revealing increased T2 signal involving one or both temporal lobes in most cases. [ 22 ] [ 14 ] Serial MRI in LE starts as an acute disease with uni- or bilateral swollen temporomesial structures that are hyperintense on fluid attenuation inversion recovery and ...
In 1825, Bouchet and Cazauvieilh described palpable firmness and atrophy of the uncus and medial temporal lobe of brains from epileptic and non-epileptic individuals. [4]: 565 In 1880, Wilhelm Sommer investigated 90 brains and described the classical Ammon's horn sclerosis pattern, severe neuronal cell loss in hippocampal subfield cornum Ammonis 1 (CA1) and some neuronal cell loss in ...
Axial fluid-attenuated inversion recovery MRI image demonstrating tumor-related infiltration involving both temporal lobes (Short arrow), and the substantia nigra ...
The lobes of the brain most likely to be injured are the frontal and temporal lobes. [19] Other common locations for DAI include the white matter in the cerebral cortex , the superior cerebral peduncles , [ 16 ] basal ganglia , thalamus , and deep hemispheric nuclei.
Brain MRI shows T2 and FLAIR hyperintensities, particularly in the medial temporal lobe. Lesions in the brain cortex or subcortex, sometimes with demyelination, may also be found. Cerebrospinal fluid (CSF) examination may show pleocytosis and oligoclonal bands. [11] [1]