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Antibiotic in case of a sudden onset of symptoms in less than 24 hours. [ 8 ] Periapical periodontitis or apical periodontitis [ 9 ] ( AP ) is an acute or chronic inflammatory lesion around the apex of a tooth root , most commonly caused by bacterial invasion of the pulp of the tooth . [ 10 ]
After endodontic therapy has been executed, or re-executed, successfully, and the canals can no longer provide a nutrient-rich habitat for microbes, [31] the issue of bone healing comes into focus. Ostensibly, then, for regeneration to occur, the root canal system must have been decontaminated and further access to microbial invasion must be ...
Root canal treatment should be performed on the tooth if it is determined that previous therapy was unsuccessful. Removal of the necrotic pulp and the inflamed tissue as well as proper sealing of the canals and an appropriately fitting crown will allow the tooth to heal under uninfected conditions.
A degree of dentin sensitivity is normal, but pain is not usually experienced in everyday activities like drinking a cooled drink. Therefore, although the terms dentin sensitivity and sensitive dentin are used interchangeably to refer to dental hypersensitivity, [3] the latter term is the more accurate.
Underneath the cementum and enamel layers, dentin forms the bulk of the tooth and surrounds the pulp system. The part of the pulp inside the crown is the pulp chamber, and the central soft tissue nutrient canals within each root are root canals, exiting through one or more holes at the root end (apical foramen/foramina).
No clinical symptoms. Pulp will respond to sensitiviity tests as a healthy pulp would. [5] [7] Treatments include root canal or tooth extraction. In endodontic therapy, removal of the inflamed pulp relieves the pain. The empty root canal system is then obturated with gutta-percha (rubber material that acts as a pressure/pain reliever). [20]
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Chronic periodontitis is initiated by Gram-negative tooth-associated microbial biofilms that elicit a host response, which results in bone and soft tissue destruction. In response to endotoxin derived from periodontal pathogens, several osteoclast-related mediators target the destruction of alveolar bone and supporting connective tissue such as the periodontal ligament.