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Smell as evidence of disease has been long used, dating back to Hippocrates around 400 years BCE. [1] It is still employed with a focus on volatile organic compounds (VOCs) found in body odor. [ 2 ] VOCs are carbon-based molecular groups having a low molecular weight, secreted during cells' metabolic processes. [ 3 ]
Body odor or body odour (BO) is present in all animals and its intensity can be influenced by many factors (behavioral patterns, survival strategies). Body odor has a strong genetic basis, but can also be strongly influenced by various factors, such as sex, diet, health, and medication. [ 1 ]
Body odor encompasses axillary (underarm) odor and foot odor. [1] It is caused by a combination of sweat gland secretions and normal skin microflora. [1] In addition, androstane steroids and the ABCC11 transporter are essential for most axillary odor. [1] [2] Body odor is a complex phenomenon, with numerous compounds and catalysts involved in ...
Body odours in general are thought to play an important role in mate selection in humans, [40] and unpleasant odour may signal disease, and hence a potentially unwise choice of mate. Although reports of bad breath are found in the earliest medical writings known, the social stigma has likely changed over time, possibly partly due to ...
Sweat and body odor are typically thought to go hand in hand, but experts say it's a little more complicated than that. Sweat alone doesn't have a smell, according to Harvard Health.
Defects in SELENBP1 gene which leads to accumulation of methanethiol and a cabbage like body odor and halitosis [4] As intestinal research progresses, it is expected that further diseases emerge that are able to explain the occurrence of all the observed types of body odor.and halitosis types and their corresponding intestinal metabolites.
Strong body odors can literally make you vomit. Chemicals in scented products can create headaches, nausea, upper respiratory problems, and skin irritation. It's time for the scent war to end on ...
A fish-like body odor can result from extreme excess consumption of TMA precursors such as choline, carnitine and betaine (usually unobtainable via regular dietary intake, it requires high levels of supplement intake). 900mg of trimethylamine, [33] 8g-20g of choline, [34] 3g of carnitine [35] or 20g betatine [36] has been known to cause ...
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