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The brachium of superior colliculus (or superior brachium) is a branch that extends laterally from the superior colliculus, and, passing to the thalamus between the pulvinar and the medial geniculate nuclei, is partly continued into an eminence called the lateral geniculate nucleus, and partly into the optic tract.
Specifically, compression or ischemic damage of the mesencephalic tectum, including the superior colliculus adjacent oculomotor (origin of cranial nerve III) and Edinger-Westphal nuclei, causing dysfunction to the motor function of the eye. Classically, it has been associated with three major groups:
It arises from the superior colliculus of the mesencephalic (midbrain) tectum, and projects to the cervical and upper thoracic spinal cord levels. [2] It mediates reflex turning of the head and upper trunk in the direction of startling sensory stimuli (visual, auditory, or skin). [2] It arises from the deep layers of the superior colliculus.
The FEF constitutes together with the supplementary eye fields (SEF), the intraparietal sulcus (IPS) and the superior colliculus (SC) one of the most important brain areas involved in the generation and control of eye movements, particularly in the direction contralateral to the frontal eye fields' location. In addition, FEF has an important ...
There are three theories for the explanation of blindsight. The first states that after damage to area V1, other branches of the optic nerve deliver visual information to the superior colliculus, pulvinar [23] [24] and several other areas, including parts of the cerebral cortex. In turn, these areas might then control the blindsight responses.
The tectopulvinar pathway is a fast-acting pathway that provides the viewer with information on the absolute spatial information of objects. The pathway plays a large role in directing visual spatial attention and is particularly responsive to novel stimuli that appear or move in peripheral vision; however, because it receives mostly magnocellular visual input, the tectopulvinar pathway is not ...
The Sprague effect is the phenomenon where homonymous hemianopia, caused by damage to the visual cortex, gets slightly better when the contralesional superior colliculus is destroyed. [1] [2] The effect is named for its discoverer, James Sprague, who observed this phenomenon in 1966 using a cat model. [3]
The optic tract is a continuation of the optic nerve that relays information from the optic chiasm to the ipsilateral lateral geniculate nucleus (LGN), pretectal nuclei, and superior colliculus. [14] The optic tract represents the first stage in the visual pathway in which visual information is transferred in a homonymous nature. [15]