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Amiodarone induced thyrotoxicosis (AIT) is a form of hyperthyroidism due to treatment with antiarrhythmic drug, amiodarone. Amiodarone induced thyroid dysfunction more commonly results in hypothyroidism , estimated to occur in 6-32% of patients, whereas hyperthyroidism from amiodarone use is estimated at 1-12%. [ 1 ]
Amiodarone has been used both in the treatment of acute life-threatening arrhythmias as well as the long-term suppression of arrhythmias. [13] Amiodarone is commonly used to treat different types of abnormal heart rhythms, such as atrial arrhythmias (supraventricular arrhythmias) and ventricular arrhythmias. [13]
Amiodarone is also safe to use in individuals with cardiomyopathy and atrial fibrillation, to maintain normal sinus rhythm. Amiodarone prolongation of the action potential is uniform over a wide range of heart rates, so this drug does not have reverse use-dependent action. Amiodarone was the first agent described in this class. [4]
Calcium channel blocker toxicity is the taking of too much of the medications known as calcium channel blockers (CCBs), either by accident or on purpose. [3] This often causes a slow heart rate and low blood pressure . [ 1 ]
Hashimoto’s disease is an autoimmune disorder that affects the thyroid gland, per the Mayo Clinic. Symptoms of the condition include fatigue, muscle aches, joint pain, problems with memory or ...
Alcohol can exacerbate the symptoms and may directly contribute to increased severity of symptoms. The reasons for toxicity vary depending on the mixture of drugs. Usually, most victims die after using two or more drugs in combination that suppress breathing, and the low blood oxygen level causes brain death. [6]
Respiratory syncytial virus, a.k.a. RSV, is a common respiratory virus that usually causes mild, cold-like symptoms, according to the Centers for Disease Control and Prevention(CDC).
The source of iodine may be from the diet, administration of iodinated contrast for medical imaging, or amiodarone (an antiarrhythmic drug). [ citation needed ] The hyperthyroidism usually develops over 2 to 12 weeks following iodine administration.