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A 12-lead ECG showing atrial fibrillation at approximately 132 beats per minute Diagram of normal sinus rhythm as seen on ECG. In atrial fibrillation the P waves, which represent depolarization of the top of the heart, are absent. Atrial fibrillation is diagnosed on an electrocardiogram (ECG/EKG). The evaluation of atrial fibrillation involves ...
Precordial leads of a 12-lead ECG from a person with short QT syndrome The mainstay of diagnosis of short QT syndrome is the 12-lead ECG. The precise QT duration used to diagnose the condition remains controversial with consensus guidelines giving cutoffs varying from 330 ms, [ 12 ] 340 ms or even 360 ms when other clinical, familial, or ...
Cardiac rhythm disturbances, such as atrial fibrillation [6] and ventricular tachycardia; [7] Inadequate coronary artery blood flow, such as myocardial ischemia [8] and myocardial infarction; [9] and electrolyte disturbances, such as hypokalemia. [10] Traditionally, "ECG" usually means a 12-lead ECG taken while lying
A 12-lead ECG showing paroxysmal supraventricular tachycardia at about 180 beats per minute. Subtypes of SVT can often be distinguished by their electrocardiogram (ECG) characteristics. Most have a narrow QRS complex , although, occasionally, electrical conduction abnormalities may produce a wide QRS complex that may mimic ventricular ...
In a person with atrial flutter, a 12-lead electrocardiogram (ECG) will demonstrate the atrial chambers of the heart contracting at a rate of 280–300 beats per minute whereas the ventricular chambers of the heart typically beat at a rate of 140–150 beats per minute. [2]
P-wave changes in left and right atrial hypertrophy. Bifid P waves (known as P mitrale) indicate left-atrial abnormality - e.g. dilatation [6] or hypertrophy. [1] If at least three different shaped P waves can be seen in a given ECG lead tracing, this implies that even if one of them arises from the SA node, at least two others are arising ...
When the atrial rhythm is irregular (as in atrial fibrillation or sinus arrhythmia) the presence of bigeminy depends on the length of the P–P interval and happens more frequently with a longer interval. As with post PVC pauses, a longer P–P interval leads to a higher chance of re-entrant circuits and thus PVCs.
[12] [18] [19] Other methods of risk stratification include observing the ventricular rate during spontaneous atrial fibrillation on a 12-lead ECG. RR intervals of less than 250 ms suggest a higher risk pathway. During exercise testing, abrupt loss of pre-excitation as heart rate increases also suggest a lower risk pathway. [8]
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