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Low pressure baroreceptors have both circulatory and renal effects, which produce changes in hormone secretion. Stimulation of these receptors causes the atria to release atrial natriuretic peptide. This hormone acts on the kidneys to increase sodium excretion, which increases urine production and thereby leads to a decrease in blood pressure.
The low-pressure baroreceptors have both circulatory and renal effects; they produce changes in hormone secretion, resulting in profound effects on the retention of salt and water; they also influence intake of salt and water. The renal effects allow the receptors to change the long-term mean pressure in the system.
These receptors also cause a renal vasodilation, resulting in increased diuresis. This decreases the blood volume, resulting in the decrease of blood pressure. [ 2 ] [ disputed – discuss ] There are two types, type A is activated by atrial wall tension in atrial contraction (during the a wave of the atrial pressure curve), type B is activated ...
There are two kinds of baroreceptors: high-pressure arterial baroreceptors and low-pressure volume receptors, both activated by the stretching of vessel walls. [7] Arterial baroreceptors are situated in the carotid sinuses and the aortic arch, while the low-pressure volume receptors, known as cardiopulmonary receptors, are in the atria ...
Increased blood volume in the right atrium leads to stretching of the atrial walls. This stretching is sensed by atrial stretch receptors [7] (which are located at the venoatrial junction [13]), causing an increase in the firing rate of group B nerve fibers (low pressure receptors). [1]
The carotid body is situated on the posterior aspect of the bifurcation of the common carotid artery. [3]The carotid body is made up of two types of cells, called glomus cells: glomus type I cells are peripheral chemoreceptors, and glomus type II cells are sustentacular supportive cells.
High pressure receptors or high pressure baroreceptors are the baroreceptors found within the aortic arch and carotid sinus. They are only sensitive to blood pressures above 60 mmHg . When these receptors are activated they elicit a depressor response; which decreases the heart rate and causes a general vasodilation .
Cushing reflex (also referred to as the vasopressor response, the Cushing effect, the Cushing reaction, the Cushing phenomenon, the Cushing response, or Cushing's Law) is a physiological nervous system response to increased intracranial pressure (ICP) that results in Cushing's triad of increased blood pressure, irregular breathing, and bradycardia. [1]