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Large placental infarcts are associated with vascular abnormalities, e.g. hypertrophic decidual vasculopathy, as seen in hypertension. [1] Very large infarcts lead to placental insufficiency and may result in fetal death. Placental infarcts are generally detected after birth, although using ultrasound may be a way to notice infarcts prenatally.
Which trigger pro-coagulator molecules to be released into the blood stream causing action of the coagulator cascade, eventually leading to placental infarction. [5] Risk factors such as diabetes, chronic blood pressure and multiple pregnancies can increase the risk of developing placental disease. [ 3 ]
Histopathology of placenta with increased syncytial knotting of chorionic villi, with two knots pointed out. The following characteristics of placentas have been said to be associated with placental insufficiency, however all of them occur in normal healthy placentas and full term healthy births, so none of them can be used to accurately diagnose placental insufficiency: [citation needed]
Alkaline phosphatase, placental type is a membrane-bound glycosylated dimeric enzyme, also referred to as the heat-stable form, that is expressed primarily in the placenta, although it is closely related to the intestinal form of the enzyme as well as to the placental-like form.
Placental disorders associated with perinatal stroke range from anatomical (site or degree of implantation) such as placenta previa [27] to placenta-maternal effects (fetal erythroblastosis). [ 28 ] Infections like chorioamnionitis cause an infection in the maternal blood, commonly leading to premature birth and the newborn experiencing brain ...
It shares symptoms with maternal floor infarction. In mothers who have already experienced it, there is a high risk of recurrence. Recurrence may be around 18%, [11] although in cases where miscarriage occur in the first trimester, it may be as high as 50%. [10]
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The pregnancy loss rate in pregnancies with confined placental mosaicism, diagnosed by chorionic villus sampling, is higher than among pregnancies without placental mosaicism. It may be that sometimes the presence of significant numbers of abnormal cells in the placenta interferes with proper placental function.