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Most cases occur during infancy when the mortality rates are at their highest. Vein of Galen malformations are a relatively unknown condition, attributed to the rareness of the malformations. Therefore, when a child is diagnosed with a faulty Great Cerebral Vein of Galen, most parents know little to nothing about what they are dealing with.
Management of cranial venous outflow obstruction involves treating the underlying cause, if identifiable, and managing the symptoms. This can include medication to reduce intracranial pressure, anticoagulation therapy to prevent thrombosis, and in some cases, surgical intervention to restore normal venous drainage. [6] [11]
The deep venous system is primarily composed of traditional veins inside the deep structures of the brain, which join behind the midbrain to form the great cerebral vein (vein of Galen). This vein merges with the inferior sagittal sinus to form the straight sinus which then joins the superficial venous system mentioned above at the confluence ...
The most frequently observed problems related to a cerebral arteriovenous malformation (AVM) are headaches and seizures, cranial nerve afflictions including pinched nerve and palsy, [2] [3] backaches, neckaches, and nausea from coagulated blood that has made its way down to be dissolved in the cerebrospinal fluid.
A developmental venous anomaly (DVA, formerly known as venous angioma) is a congenital variant of the cerebral venous drainage. On imaging it is seen as a number of small deep parenchymal veins converging toward a larger collecting vein.
Absence of the great cerebral vein is a congenital disorder.The deep cerebral veins of the brain normally drain through the great cerebral vein. In its absence, the veins from the diencephalon and the basal ganglia drain laterally into the transverse sinus instead of conjoining in the midline through the Galenic drainage system. [8]
The superior thalamostriate vein or terminal vein commences in the groove between the corpus striatum and thalamus, receives numerous veins from both of these parts, and unites behind the crus of the fornix with the superior choroid vein to form each of the internal cerebral veins.
Symptoms: Pain, weakness, loss of muscle at the base of the thumb, swelling, paleness, bluish coloration [1] [2] Usual onset: 20 to 50 years of age [1] Types: Neurogenic, venous, arterial [1] Causes: Compression of the nerves, arteries, or veins in the superior thoracic aperture (thoracic outlet), the passageway from the lower neck to the ...