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The most common and simple treatment is the construction of a specially made acrylic prosthesis that covers the biting surfaces of the teeth and protects the cheek, tongue, and labial mucosa (an occlusal splint). This is either employed in the short term as a habit-breaking intention or more permanently (e.g., wearing the prosthesis each night ...
Alveolar mucosa, the lining between the buccal and labial mucosae. It is a brighter red, smooth, and shiny with many blood vessels, and is not connected to underlying tissue by rete pegs. [6] Buccal mucosa, the inside lining of the cheeks; part of the lining mucosa. Labial mucosa, the inside lining of the lips; part of the lining mucosa. [7]
Later during the development of the jaws, permanent molars arise directly from the distal extension of the dental lamina. The vestibular lamina is the thickening of oral epithelium in a facial [6] or buccal direction from the dental lamina. Meanwhile, a cleft will form a groove that becomes the area of the mucobuccal or mucolabial fold in the ...
In dentistry, the linea alba (from Latin 'white line') [1] is a horizontal streak on the buccal mucosa (inner surface of the cheek), level with the occlusion (biting plane). ). It usually extends from the commissure to the posterior teeth, and can extend to the inner lip mucosa and corners of the mo
Smokeless tobacco keratosis (STK) [4] is a condition which develops on the oral mucosa (the lining of the mouth) in response to smokeless tobacco use. Generally it appears as a white patch, located at the point where the tobacco is held in the mouth. The condition usually disappears once the tobacco habit is stopped.
White sponge nevus (WSN) is an extremely rare autosomal dominant [4] condition of the oral mucosa (the mucous membrane lining of the mouth). It is caused by one or more mutations in genes coding for keratin, which causes a defect in the normal process of keratinization of the mucosa.
Leukoedema lesions disappear when the mucosa is stretched, which helps to differentiate it from other white lesions in the mouth. [2] The differential diagnosis is with leukoplakia , oral candidiasis , oral lichen planus , white sponge nevus , morsicatio buccarum , [ 3 ] hereditary benign intraepithelial dyskeratosis and dyskeratosis congenita.
This defensive thickening is a primary function of epithelium. Histopathologically, numerous papillary projections are usually covered by hyperplastic stratified squamous epithelium with or without chronic inflammation. [22] Pseudoepitheliomatous appearance can be seen In advanced cases, this hyperplasia is pseudoepitheliomatous in appearance. [23]