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Immunofluorescence pattern of SS-A and SS-B antibodies. Produced using serum from a patient on HEp-20-10 cells with a FITC conjugate. Anti-SSA autoantibodies (anti–Sjögren's-syndrome-related antigen A autoantibodies, also called anti-Ro, or similar names including anti-SSA/Ro, anti-Ro/SSA, anti–SS-A/Ro, and anti-Ro/SS-A) are a type of anti-nuclear autoantibodies that are associated with ...
Leading cause of death (2016) (world) The following is a list of the causes of human deaths worldwide for different years arranged by their associated mortality rates. In 2002, there were about 57 million deaths.
Human infectious diseases may be characterized by their case fatality rate (CFR), the proportion of people diagnosed with a disease who die from it (cf. mortality rate).It should not be confused with the infection fatality rate (IFR), the estimated proportion of people infected by a disease-causing agent, including asymptomatic and undiagnosed infections, who die from the disease.
Although the autoimmune CHB has a relatively high mortality and morbidity rates, the chance of kids from -mothers positive to anti-Ro\SSA and/or anti-La\SSB antibodies- to suffer from CHB is only around 1-5%, [9] [7] [10] which suggests the existence of other factors to influence the disease such as genetic and environmental factors. [4]
The cause is generally either paraneoplastic syndrome or idiopathic. In idiopathic AAG, the body's own immune system targets a receptor in the autonomic ganglia, which is part of a peripheral nerve fiber. If the AAG is paraneoplastic, they have a form of cancer, and their immune system has produced paraneoplastic antibodies in response to the ...
Anti-mitochondrial antibodies - rare except for overlap syndromes with primary biliary cholangitis; Anti-soluble liver antigen/liver pancreas antibody antigen - 20% of people; Anti-double stranded DNA - 30% of people; Atypical perinuclear anti-neutrophil cytoplasmic antibodies (p-ANCA) Type 2 autoimmune hepatitis. Positive antibodies include ...
The exact cause is unclear. A combination of consistent findings on physical examination, the presence of anti HMG-CoA reductase antibodies in a person with myopathy, evidence of muscle breakdown, and muscle biopsy diagnose SAAM. [3] Treatment involves stopping the associated statin medication and taking medication to suppress the immune system.
Another genetic feature of MCTD is the presence of anti-RNP antibodies. However, these antibodies are not present in all patients. [56] Genome-wide association studies have revealed that there are parts of a patient’s genetic material which cause production of these anti-RNP antibody. The mechanism is not yet thoroughly defined. [57] [10]