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A late effect can be caused directly by the earlier condition, or indirectly by the treatment for the earlier condition. Some late effects can occur decades later. Historically, late effects have been very difficult to connect with their causes, but as survival and life span have increased and "follow up" has become standard practice, these ...
437.1 Other generalized ischemic cerebrovascular disease; 437.2 Hypertensive encephalopathy; 437.3 Cerebral aneurysm nonruptured; 437.4 Cerebral arteritis; 437.5 Moyamoya disease; 437.6 Nonpyogenic thrombosis of intracranial venous sinus; 437.7 Transient global amnesia; 438 Late effects of cerebrovascular disease. 438.0 Cognitive deficits; 438. ...
Acquired cerebrovascular diseases are those that are obtained throughout a person's life that may be preventable by controlling risk factors. The incidence of cerebrovascular disease increases as an individual ages. [21] Causes of acquired cerebrovascular disease include atherosclerosis, embolism, aneurysms, and arterial dissections. [9]
Approximately 10% of these silent strokes are silent lacunar infarctions. While dubbed "silent" due to the immediate lack of classic stroke symptoms, SLIs can cause damage to the surrounding brain tissue and can affect various aspects of a person's mood, personality, and cognitive functioning .
Left sided cerebellar stroke due to occlusion of a vertebral artery. Cerebellar strokes account for only 2-3% of the 600,000 strokes that occur each year in the United States. [3] They are far less common than strokes which occur in the cerebral hemispheres.
Macrovascular disease is a disease of any large (macro) blood vessels in the body. It is a disease of the large blood vessels, including the coronary arteries, the aorta, and the sizable arteries in the brain and in the limbs.
In addition, the pressure may lead to a loss of blood supply to affected tissue with resulting infarction, and the blood released by brain hemorrhage appears to have direct toxic effects on brain tissue and vasculature. [53] [75] Inflammation contributes to the secondary brain injury after hemorrhage. [75]
Cerebral atherosclerosis is a type of atherosclerosis where build-up of plaque in the blood vessels of the brain occurs. Some of the main components of the plaques are connective tissue, extracellular matrix, including collagen, proteoglycans, fibronectin, and elastic fibers; crystalline cholesterol, cholesteryl esters, and phospholipids; cells such as monocyte derived macrophages, T ...