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Hip dysplasia is an abnormality of the hip joint where the socket portion does not fully cover the ball portion, resulting in an increased risk for joint dislocation. [1] Hip dysplasia may occur at birth or develop in early life. [1] Regardless, it does not typically produce symptoms in babies less than a year old. [3]
There are typically four classes (or types) of PFFD, ranging from class A to class D, as detailed by Aitken. [4] [5]Type A — The femur bone is slightly shorter on the proximal end (near the hip), and the femoral head (the ball of the thigh bone that goes into the hip socket) may not be solid enough to be seen on X-rays at birth, but later hardens (ossifies).
Hip dysplasia is a congenital condition in which the hip is deformed in a way that decreases the congruency between the head of the femur and the acetabulum of the pelvis. [22] Bony congruence is a stabilizing factor to the hip joint, so the decrease in this conferred by hip dysplasia makes one more susceptible to dislocation. [22]
The hip joint is classified as a ball and socket joint. This type of synovial joint allows for multidirectional movement and rotation. There are two bones that make up the hip joint and create an articulation between the femur and pelvis. This articulation connects the axial skeleton with the lower extremity.
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Protrusio acetabuli is an uncommon defect of the acetabulum, the socket that receives the femoral head to make the hip joint. The hip bone of the pelvic bone/girdle is composed of three bones, the ilium, the ischium and the pubis. In protrusio deformity, there is medial displacement of the femoral head in that the medial aspect of the femoral ...
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