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Rhythm strip of sinus bradycardia at 50 bpm. Sinus bradycardia is commonly seen in normal healthy persons and athletes in the absence of pathophysiological diseases or conditions. [1] Different factors or etiologies could lead to the dysfunction of the sinus node, causing a malformation or prolongation of the impulse. In terms of ...
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This image is a derivative work of the following images: File:Sinusbradylead2.JPG licensed with Cc-by-sa-3.0, GFDL 2010-09-26T22:18:43Z Jmh649 1300x489 (191900 Bytes) {{Information |Description={{en|1=Sinus bradycardia as seen in lead 2.
Bradycardia, also called bradyarrhythmia, is a resting heart rate under 60 beats per minute (BPM). [1] While bradycardia can result from various pathologic processes, it is commonly a physiologic response to cardiovascular conditioning or due to asymptomatic type 1 atrioventricular block.
A sinus rhythm is any cardiac rhythm in which depolarisation of the cardiac muscle begins at the sinus node. [1] It is necessary, but not sufficient, for normal electrical activity within the heart. [2] On the electrocardiogram (ECG), a sinus rhythm is characterised by the presence of P waves that are normal in morphology. [2]
The ECG can detect sinus bradycardia, a resting heart rate of fewer than 60 beats per minute. This is often accompanied by sinus arrhythmia. The pulse of a person with athlete's heart can sometimes be irregular while at rest, but usually returns to normal after exercise begins. [12] [13] [14]
The constant interval between the sinus beat and PVC suggests a reentrant etiology rather than spontaneous automaticity of the ventricle. [3] Premature atrial contractions by contrast do not have a compensatory pause, since they reset the sinus node, but atrial or supraventricular bigeminy can occur. If the PACs are very premature, the ...
This most commonly occurs in the setting of a sinus bradycardia. [5] Accelerated idioventricular rhythm is the most common reperfusion arrhythmia in humans. However, ventricular tachycardia and ventricular fibrillation remain the most important causes of sudden death following spontaneous restoration of antegrade flow. [6]